The lost ability to digest milk is a common problem among the two-thirds of the population of the world which are above childhood phase and according to science, after weaning, almost all the mammals lose the ability to digest milk.
This lactose gets broken into two small components sugar with the help of an enzyme named as lactase as it cannot be easily absorbed in the tract of the intestine. LCT, the lactase producing gene gets declined after childhood.
According to the recent study, few evidences are found, according to which the decline of LCT did not happen due to any changes made in genes, the true fact is; due to the chemical modification done in DNA, the lactase digesting gene gets switched off. These modifications are termed as epigenetic.
The new study done on lactose intolerance disorder gives information regarding its development in tract with age and trauma.
A scientist revealed his experience, where he had seen that the person lost his lactose digestive ability at 45 years of age. In the same way, it is possible with other humans also that they can digest lactose after a long period of time of their life. Some people regain this ability after treatment and some become permanent intolerant.
It is said through the study that this problem can be caused in individuals due to trauma that can result in turning of lactase digesting gene during childhood. The study states that for understanding these alterations, more researches should be done.
According to a recent study, the persistence rate of lactase enzyme in adulthood is 35% of the world’s adult population, and these ranges are highly different in ethnic groups.
In the United States, the rate of people who are lactose-tolerant is up to 64% and this proportion reflects the rate of the population belonging to the ethnic group.
It is only revealed in recent studies that adults can also have the ability to digest lactase. SNPs are genetic changes that convey the persistence of lactase.
Scientists are continuously trying to find out the reasons for changes that occurred in genes of humans which cause lactase-intolerance.